Overexpression of extracellular superoxide dismutase protects against brain injury induced by chronic hypoxia

Keyphrases

• Brain Injury 100%
• Overexpression 100%
• Chronic Hypoxia 100%
• Extracellular Superoxide Dismutase (EC-SOD) 100%
• Hypoxia 40%
• Hippocampus 40%
• Room Air 40%
• Brain Metabolism 20%
• Positron Emission Tomography 20%
• Blood Vessels 20%
• Wild Mice 20%
• Synaptic Plasticity 20%
• Hippocampal Slice 20%
• Cerebellum 20%
• Brain Oxygenation 20%
• Apoptotic Cells 20%
• Severely Affected 20%
• Transgenic Mice 20%
• Hypothalamus 20%
• Oxidative Stress Markers 20%
• Transgenic Animals 20%
• WT Mice 20%
• Iba1 20%
• Functional State 20%
• Therapeutic Role 20%
• No Significant Difference 20%
• Cell Damage 20%
• Caspase-3 20%
• Histopathological Study 20%
• Electrophysiological Techniques 20%
• 18F-fluorodeoxyglucose (18F-FDG) 20%
• SOD Activity 20%
• P-AMPK 20%

Neuroscience

• Extracellular Superoxide Dismutase 100%
• Hippocampus 40%
• Brain Metabolism 20%
• Positron Emission Tomography 20%
• Cell Damage 20%
• Oxidative Stress 20%
• Synaptic Plasticity 20%
• Caspase 3 20%
• Cerebellum 20%
• Brain Injury 20%
• Hypothalamus 20%
• Electrophysiological Technique 20%