Vitamin E deficiency and immune dysfunction in retrovirus-infected C57BL/6 mice are prevented by T-cell receptor peptide treatment

Keyphrases

• T Cell Receptor 100%
• Peptide Drugs 100%
• C57BL 100%
• Retrovirus 100%
• Immune Dysfunction 100%
• Vitamin E Deficiency 100%
• Vitamin E 66%
• AIDS/HIV 50%
• Retroviral Infection 50%
• Oxidative Damage 33%
• Lipid Peroxidation 33%
• Tissue Lipids 33%
• Liver 16%
• Immune Function 16%
• T Cells 16%
• Interleukin-6 16%
• Post-infection 16%
• T Cell Subsets 16%
• Interleukin-2 16%
• T Helper 1 16%
• Lipid Loss 16%
• Lipid Peroxides 16%
• Splenocytes 16%
• Th2 Cytokines 16%
• LP-BM5 16%
• Superantigen 16%

Pharmacology, Toxicology and Pharmaceutical Science

• Receptor 100%
• C57BL 6 Mouse 100%
• T Lymphocyte Receptor 100%
• Retrovirus 100%
• Alpha Tocopherol Deficiency 100%
• Alpha Tocopherol 66%
• Retrovirus Infection 50%
• Cytokine 33%
• Murine Acquired Immunodeficiency Syndrome 33%
• Mouse 16%
• Infection 16%
• Acquired Immune Deficiency Syndrome 16%
• Interleukin 2 16%
• Lipid Peroxide 16%
• Superantigen 16%

Immunology and Microbiology

• T Cell Receptor 100%
• C57BL 6 Mouse 100%
• Vitamin E 100%
• Immune Disorder 100%
• Retrovirus 100%
• Acquired Immune Deficiency Syndrome 42%
• Retrovirus Infection 42%
• Cytokine 28%
• Lipid Peroxidation 28%
• Mouse 14%
• T Cell 14%
• Interleukin-6 14%
• Secretion (Process) 14%
• T Cell Subset 14%
• Interleukin 2 14%
• Splenocyte 14%
• Superantigen 14%